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Occupational hazards, including production of inorganic dust and dust. Cause pulmonary fibrosis are mainly inorganic dust, silicon dioxide dust (dust free silica) and silicate dust (asbestos, talc, mica and kaolin dust, etc.).

If you are a worker, in a short time of exposure to high concentrations of silicon dioxide, may be acute silicosis. There are many visible double lung nodules, while visible septal thickening and ground-glass opacity. Pneumoconiosis patients both nature changes, but also changes in alveolar protein calm psychosis autoimmunity.

There are some workers not only have pneumoconiosis, and some may airway inflammation, a narrow form of emphysema, bulla. If coal dust exposure and smoking merger will increase the patient's condition.

Epidemiological survey shows that China COPD patients, about one-fifth of patients with occupational dust exposure. These inorganic dust exposure include dust, organic dust, as well as a number of irritating chemicals.

Dust and cancer has a relevance. Now more are mentioned asbestos cause cancer problem. But in fact, the current international organizations silicon dioxide is classified as a carcinogen.

Pneumoconiosis complications and complications include: Respiratory failure; pulmonary hypertension, chronic pulmonary heart disease; pneumothorax; chronic obstructive pulmonary disease, bronchial asthma; bronchial - lung infection; dust tuberculosis, bronchial - pulmonary fungal disease; venous thrombosis embolism; lung cancer. These complications and comorbidities cover almost all professional respiratory medicine, including airway, allergy, blood vessels, infections and tumors.

 

 

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